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Hi, Welcome to Just Answer. I'm Chris, a Vet based in the UK. I am reviewing your question and will get back to you asap with a response
Could you tell me more about their symptoms and why you think that selinium poisoning may be likely? Thanks
OK thanks for that. So testing of hair samples can be useful, however it does not tell you when the selenium toxicity occurred or if it is still ongoing (hair samples reflect toxicity occurring over months to years). Blood samples would tell you if the high selenium intake is still happening now. The most likely cause is high levels in the local soil where the horses graze or where their feed is grown (if fed hay etc). Soil sample or feed sample testing can also be conducted. Treatment really only involves decreasing the selenium intake and waiting for signs to resolve, this could however take several months until new hair and hoof develops.
I hope that answers your question? Please let me know if you need any further help or advice. Chris
OK that's good, it suggests whatever caused the toxicity may have now passed.
I agree there is rotation of the pedal bone on your xray, so if comparible clinical signs are present (increased heat and digital pulses), then laminitis is likely.
Management of laminitis is primarily dietary (reduce feed intake/quality/reduce concentrates), alongside remedial farriery (frog supports) and of course pain relief (flunexin, phenylbutazone). Persistently affected horses should be tested for equine Cushings disease via blood samples
Again possibly a dietary issue, have you tried a biotin supplement for them?
Yes they are fairly common. They are sometimes described as 'sand cracks' see https://vcahospitals.com/know-your-pet/hoof-wall-cracks-in-horses
Remedial farriery and dietary supplementation is usually the treatment of choice (alongside pain relief)
Do the coronary band lesions ooze at all?
OK good. In which case it is likely to be linked to the issue with selenium. Other causes include coronary band dystrophy, pemphigus and certain types of mange (choroptic) . If the lesions are not improving or seem to worsen with time despite appropriate diet/supplements and farriery then biopsy of the coronary band for histopathology (if available) would be the next step to consider.
At the coronet band if an immune mediated disorder was present
I think it is unlikely for the whole hoof to Slough if selenium toxicity (which is now hopefully resolving) is the cause
If they are lame then box rest would be advisible until the lameness is controlled, it is a little unclear here how much the lameness is due to laminitis and how much is due to pain from the hoof cracking - it is possibly an element of both unfortunately
Consider pain relief if very lame - flunexin or bute